Tuesday, March 18, 2014

Depression and Anti-depressants Project


Depression and anti-depressants

Depression affects 19 million people a year. One of the ways doctors treat depression is through prescribing anti-depressants. Anti-depressants are the 2nd most prescribed drug in America.

Symptoms of depression
  • Persistent sad, anxious or "empty" mood
  • Sleeping too much or too little, middle of the night or early morning waking
  • Reduced appetite and weight loss, or increased appetite and weight gain
  • Loss of pleasure and interest in activities once enjoyed, including sex
  • Restlessness, irritability
  • Persistent physical symptoms that do not respond to treatment (such as chronic pain or digestive disorders)
  • Difficulty concentrating, remembering or making decisions
  • Fatigue or loss of energy
  • Feeling guilty, hopeless or worthless
  • Thoughts of suicide or death

Causes of depression
·       Biological - People with depression typically have too little or too much of certain brain chemicals, called "neurotransmitters."  Changes in these brain chemicals may cause or contribute to clinical depression.
·       Cognitive - People with negative thinking patterns and low self-esteem are more likely to develop clinical depression.
·       Gender - Women experience clinical depression at a rate that is nearly twice that of men.[3]  While the reasons for this are still unclear, they may include the hormonal changes women go through during menstruation, pregnancy, childbirth and menopause.  Other reasons may include the stress caused by the multiple responsibilities that women have.
·       Co-occurrence - Clinical depression is more likely to occur along with certain illnesses, such as heart disease, cancer, Parkinson's disease, diabetes, Alzheimer's disease and hormonal disorders.
·       Medications - Side effects of some medications can bring about depression.
·       Genetic - A family history of clinical depression increases the risk for developing the illness.
·       Situational - Difficult life events, including divorce, financial problems or the death of a loved one can contribute to clinical depression.


Side effects of anti-depressant medication
- agitation
- sexual dysfunction
- can be associated with movement disorders such as akathisia, Parkinson’s disease, dystonia (acute rigidity), dyskinesia (abnormal involuntary choreic movements) and tardive dyskiniesia
- upset stomach
- nausea
- HA
- fatigue
- tremor
- nervousness
- dry mouth
- insomnia
- weight gain
- improper bone development
- improper brain development
- gastrointestinal bleeding
- amotivational syndrome


 Doctors are prescribing additional medications to help control the side effects of the anti-depressant. For example:
            - ex. Prozac can cause agitation so a sedative might be prescribed with it
            - ex. If gastrointestinal bleeding occurs docs are prescribing acid-inhibiting drugs
            - these new drugs can cause side effects leading to the need for other drugs


Research

FDA admitted that “two to three children out of every hundred” could be expected to develop suicidal thoughts or actions as a result of antidepressant therapy (Harris 2004). The risk of suicide events for children receiving SSRIs has been three times higher than placebo. 2-4 times higher chance in adults then with placebo

In a study designed to investigate the anatomic effects of serotonergenic compounds, researchers at Thomas Jefferson University found that high-dose, short-term exposure to SSRIs in rats was sufficient to produce swelling and kinking in the serotonin nerve fibers (Kalia 2000). These side effect reversed after prolonged recovery period.

Research performed by a different team of investigators demonstrated a reduction in dendritic length and dendritic spine density, and in contrast to the previous study, these changes did not reverse even after a prolonged recovery period. The results were interpreted to suggest that chronic exposure to SSRIs may arrest the normal development of neurons.

Andrews et al. (2011), like El-Mallakh et al. (2011), stress what I had first described as compensatory mechanisms. SSRI antidepressants block the removal of serotonin from the synapses between neurons, in effect trying to flood these synapses with serotonin. Many studies confirm that the brain attempts to compensate for the impact of the SSRIs by reducing the brain's capacity to respond to serotonin. This leads to a loss of serotonin receptors that can reach 60 percent. Blockade of serotonin reuptake causes a potentially harmful adaptive response in the form of a persistent hypertrophy of the reuptake mechanism. Additional studies show persistent biochemical changes in the brain following exposure to SSRI antidepressants. 

In a meta-analysis of 46 studies, Andrews et al. (2011) found the relapse rate for antidepressant-treated patients (44.6 percent) was much higher than for placebo-treated patients (24.7 percent). Andrews also found that the more potent antidepressants caused an increased risk of relapse on drug discontinuation. A 2010 Minnesota evaluation of patient care in the state found that only 4.5 percent of more than 20,000 patients were in remission at 12 months, indicating that they had become chronically afflicted with depression during and probably as a result of their treatment.

Although many negative opinions have been stated, some people do have a serious chemical imbalance and need anti-depressants. In some studies anti-depressants did show to improve people’s lives. Anti-depressants have also not been linked to any serious diseases like cancer and heart disease. In my opinion people should seek counseling if they are having problems with depression. Working out also shows to have positive results with improving depression. I think if a person is trying to get help for depression without taking anti-depressants and their depression is not getting better then trying anti-depressants might be a good idea.

 Sources

http://www.youtube.com/watch?v=sc-4zhqViQ4 




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